mark klempner is a known liar and fraud who should be stripped of his medical degree

eugeneshapiroisapig · medicine forum Guru Joined: 24 Mar 2005 Posts: 2108

following klempner's filthy 2001 propagandistic pile of excrement, he
appeared to make a distinction between "treatment-resistant
neuroborreliosis" and "chronic post treatment lyme disease". in 2002,
in a commentary which appeared in the corrupt durland fish's pathetic
shitty journal, klempner writes:

'Unlike treatment resistant neuroborreliosis, where there appears to be
an active inflammatory component, there is little evidence in patients
with "post-Lyme syndrome".

One wonders, then, how exactly klempner was able to determine that
there is an active inflammatory component in neuroborreliosis,
treatment resistant or not, since it is well known that even untreated
late neuroborreliosis/lyme encephalopathy can present with completely
normal CSF results. Why is this? Because there is a huge and obvious
difference between acute meningitis, which occurs in early LD and for
which CSF findings such as elevated protein are frequent, and diffuse,
disseminated late neuroborreliosis which occurs years after the initial
infection. Culture and PCR of CSF in even late stage untreated cases is
also difficult, a fact which many including steere acknowledge. Since
the lack of CSF findings cannot be used to justify diagnostic exclusion
in these untreated cases, how can the lack of diagnostic findings in
treated cases be used as "proof" of the efficacy of treatment?

So how exactly does klempner justify this sweeping assertion, that
postlyme syndrome is different from treatment resistant
neuroborreliosis? Where is his 'evidence'? Does it lay somewhere within
the discarded pile of patients, those presenting with mood disorders
who were reportedly excluded from his study? Does it lay somewhere
buried deep beneath the fact, widely accepted in neurology and
neuropsychiatry for decades, that subacute or chronic organic brain
disease almost always presents with depressive, anxious, or even
psychotic features?

And what should we make of recent reports and indications which hint
that related borrelia specifically utilize the CNS as a host reservoir,
and that the CNS immune system does not react in the same way to
borrelia as the rest of the body's immune system?

What does klempner know, and when did he know it? Or does he just think
he knows it?

Can we believe anything this man says? Why was he made the lead editor
for ID articles for the NEJM following his 2001 paper? Why was he given
control of a $100 million+ biodefense laboratory? Does our country
really need men of his ilk in such positions of power?

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