Yale and Plum Island Mycoplasma- stealth fungal infections that turn off the immune system.

kathleen · medicine forum Guru Joined: 24 Mar 2005 Posts: 2619

From: Kathleen Dickson <kmdickson0308@yahoo.com>
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Subject: Yale and Plum Island Mycoplasma- stealth fungal infections
that turn off the immune system.

Date: Saturday, May 27, 2006 12:57:26 [View Source]

As you can see, I quote the published science:

http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=PureSearch&db=pubmed&details_term=6190898%5BUID%5D
1: J Hyg (Lond). 1983 Jun;90(3):441-9.

Immunogenic variation among the so-called LC
strains of Mycoplasma mycoides subspecies mycoides.

Smith GR, Oliphant JC.

Much evidence of immunogenic heterogeneity among
the LC strains of Mycoplasma mycoides ssp. mycoides
emerged from cross-immunization and
-hyper-immunization experiments in mice in which three
LC strains ***(Vom/Plum Island, 74/2488,*** and
Mankefar 2833) were used for challenge purposes. All
heterologous LC-strain vaccines cross-immunized
against the three challenge strains, but protection
was usually only 'partial', i.e. significantly less
than that given by homologous vaccine.
Cross-hyperimmunization with all heterologous LC but
not SC strains produced protection against challenge
with Vom/Plum Island that was virtually 'complete',
i.e. similar to that produced by homologous vaccine.
Challenge with 74/2488 gave generally similar results;
but against Mankefar 2833 six heterologous LC vaccines
gave complete protection and six did not. Vaccines
prepared from the Smith (1423) strain of M. mycoides
ssp. capri gave some protection against Vom/Plum
Island but none against 74/2488 or Mankefar 2833. The
cross-immunizing ability of three further M. mycoides
ssp. capri strains appeared to resemble that of Smith
(1423). In a cross-hyperimmunization experiment,
vaccines prepared from SC strains of M. mycoides ssp.
mycoides varied greatly in their ability to protect
against challenge with strains 74/2488 and Mankefar
2833.
PMID: 6190898 [PubMed - indexed for MEDLINE]
==========
Yale's Vector-Pathogen Competence Studies:
DURLAND FISH- the one who conpired to send the Lyme
Disease Foundation "a bogus article."
http://actionlyme.org/TICK_BITE_CONSPIRACY.htm

http://jvi.asm.org/cgi/content/full/72/3/1711?view=long&pmid=9499019

J Virol, March 1998, p. 1711-1724, Vol. 72, No. 3
0022-538X/98/$04.00+0
Copyright © 1998, American Society for Microbiology.
All rights reserved.
African Swine Fever Virus Infection in the Argasid
Host, Ornithodoros porcinus porcinus
S. B. Kleiboeker,1 T. G. Burrage,1 G. A. Scoles,1,2 D.
Fish,2 and D. L. Rock1,*

Plum Island Animal Disease Center, Agricultural
Research Service, U.S. Department of Agriculture,
Greenport, New York 11944,1 and Department of
Epidemiology and Public Health, Yale University School
of Medicine, New Haven, Connecticut 065202

Received 3 October 1997/Accepted 24 November 1997
ABSTRACT

The pathogenesis of African swine fever virus (ASFV)
infection in Ornithodoros porcinus porcinus was
examined in nymphal ticks infected with the ASFV
isolate Chiredzi/83/1. At times postinfection (p.i.)
ranging from 6 h to 290 days, ticks or dissected tick
tissues were titrated for virus and examined
ultrastructurally for evidence of virus replication.
The ASFV infection rate in ticks was 100% in these
experiments, and virus infection was not associated
with a significant increase in tick mortality. Initial
ASFV replication occurred in phagocytic digestive
cells of the midgut epithelium. Subsequent infection
and replication of ASFV in undifferentiated midgut
cells was observed at 15 days p.i. Generalization of
virus infection from midgut to other tick tissues
required 2 to 3 weeks and most likely involved virus
movement across the basal lamina of the midgut into
the hemocoel. Secondary sites of virus replication
included hemocytes (type I and II), connective tissue,
coxal gland, salivary gland, and reproductive tissue.
Virus replication was not observed in the nervous
tissue of the synganglion, Malpighian tubules, and
muscle. Persistent infection, characterized by active
virus replication, was observed for all involved tick
tissues. After 91 days p.i., viral titers in salivary
gland and reproductive tissue were consistently the
highest detected. Successful tick-to-pig transmission
of ASFV at 48 days p.i. correlated with high viral
titers in salivary and coxal gland tissue and their
secretions. A similar pattern of virus infection and
persistence in O. porcinus porcinus was observed for
three additional ASFV tick isolates in their
associated ticks.

=====
"let's remember that in 1991 we found that their
weapons of mass destruction programs were far further
developed than anyone knew." -- Condi Rice.
http://www.scoop.co.nz/stories/WO0605/S00421.htm
ROTFLMAO

Uh, Ms. Rice, would that be the reason Poppy Bush
(George H. W. Bush) did not finish off Saddam Hussein
in 1991? Because he found that Iraq was actually
using the bioweapons we gave them (mycoplasmal,
stealth fungal infections, etc)? And that that might
be a cause of Gulf War Illness?
http://actionlyme.org/BIOWEAPONEERS_CORIXA_YALE_TLRS.htm

And could, "Russian President Putin behaving
differently," be because he found out 9/11 and the
intelligence were being fixed around the Israeli
Realm-Securing policy, he found out who the real
terrorists were, and he might be more than a little
pissed?
---

Mr. W Coke-head Grandson of Mr. Prescott NAZI funding
Warbucks and the Walker family,
http://actionlyme.org/LYME_CORRUPTICUT.htm
knows what he can do with his "democracy" and
"freedom," and the same for the incompetent, vicious,
"Torture is okay," Worm Gonzales.

For the doubters, Lyme and LYMErix cause the same
immune suppression that mycobacteria and mycoplasma
cause:
***prolonged TLR-2 signaling in macrophages results in
down-regulation of certain critical immune functions,
such as MHC-II Ag processing.***

"Lipoproteins and lipopeptides have been identified in
a large number of microorganisms, the most prominent
ones being mycobacteria, mycoplasms, and spirochetes.
They have been found to exhibit both a strong innate
inflammatory response in the host and an enduring
adaptive immune response in mammalian hosts (16). The
strong proinflammatory capacities of lipoproteins were
first described for outer surface proteins A and B of
Borrelia burgdorferi, which are also highly
immunogenic (17) and have lately been the basis for a
Lyme disease vaccine development (1 Cool

. These compounds
exhibit an triacylated lipid anchor structure
comprising an
N-palmitoyl-S-[2,3-bis(palmitoyloxy)-(2RS)-propyl]-(R)-cysteinyl
(Pam3Cys) moiety at the N terminus (19), a feature
that was previously described for the Braun
lipoprotein from Escherichia coli (20). Because the
N-terminal Pam3Cys modification is essential for
immunoactivation caused by lipoproteins of B.
burgdorferi as well as of another spirochete,
Treponema pallidum (21), subsequent studies
investigating immune responses to spirochetes used
synthetic lipopeptides (22). The Pam3Cys moiety was
also reported to be present in cytokine-inducing
lipoproteins of Mycobacterium and Mycoplasma spp. (23,
24); thus, it can be regarded as a highly conserved
molecular motif among different classes of bacteria.
In Mycoplasma fermentans, the presence of a macrophage
stimulating lipopeptide, termed 2-kDa
macrophage-activating lipopeptide (MALP-2), was
observed, being stimulatory active at picomolar
concentrations (25). This compound, in contrast to the
predominant lipopeptide structures present in
lipoproteins of E. coli, B. burgdorferi, and
mycobacteria, lacks the N-palmitoyl group, thus
containing a diacylated (Pam2Cys) lipid anchor
structure at the N terminus. Following studies
revealed the presence of closely related compounds in
other Mycoplasma spp. (26).." from:
http://www.jimmunol.org/cgi/content/full/173/4/2683

How LYMErix caused illness- immune suppression:

"Signaling through TLR-2 by lipoproteins may
represent a double-edged sword for host responses to
chronic intracellular pathogens such as M.
tuberculosis. Short-term signaling through TLR-2
activates macrophages and initiates acute inflammation
that may help control initial infection. In contrast,
***prolonged TLR-2 signaling in macrophages results in
down-regulation of certain critical immune functions,
such as MHC-II Ag processing.*** M. tuberculosis
infects, survives, and persists in macrophages. The
ability of M. tuberculosis to survive acute
inflammation positions the bacilli to take advantage,
through secretion of lipoproteins such as LprG and
LpqH, of this down-regulation of macrophage immune
function."
http://www.jimmunol.org/cgi/content/full/173/4/2660
The Journal of Immunology, 2004, 173: 2660-2668.
Copyright © 2004 by The American Association of
Immunologists Mycobacterium tuberculosis LprG
(Rv1411c): A Novel TLR-2 Ligand That Inhibits Human
Macrophage Class II MHC Antigen Processing1 Adam
J. Gehring*, Karen M. Dobos, John T. Belisle, Clifford
V. Harding2, and W. Henry Boom2,3,*,, * Division
of Infectious Diseases, Department of Pathology, and
Tuberculosis Research Unit, Case Western Reserve
University and University Hospitals of Cleveland,
Cleveland, OH 44106; and Mycobacteria Research
Laboratories, Department of Microbiology, Immunology,
and Pathology, Colorado State University, Fort
Collins, CO 80523

That means Chronic Lyme is Chronic because it is
chronic. Says Alan Barbour:
http://patft1.uspto.gov/netacgi/nph-Parser?Sect1=PTO1&Sect2=HITOFF&d=PALL&p=1&u=%2Fnetahtml%2FPTO%2Fsrchnum.htm&r=1&f=G&l=50&s1=6,719,983.PN.&OS=PN/6,719,983&RS=PN/6,719,983

"2.1 Methods of Treatment

"An important aspect of the invention is the
recognition that Borrelia VMP-like sequences recombine
at the vls site, with the result that antigenic
variation is virtually limitless. Multiclonal
populations therefore can exist in an infected patient
so that immunological defenses are severely tested if
not totally overwhelmed."

That means the Yale/Steere/Dearborn method to diagnose
"Lyme disease" is useless.
-----
---

I hope this helps everyone understand that we have no
natural ability to be the world police, because
Americans are too stupid, evil, cowardly, immoral, and
incompetent to be anyone's guard. The war on terror
began here at home in Nov 2000.
http://web.archive.org/web/20060527094034/http://groups.google.com/group/sci.med.diseases.lyme/search?group=sci.med.diseases.lyme&q=Bush/Gore+ENERGY+&qt_g=1&searchnow=Search+this+group

Blood for oil. Dubya explained to us his plans in the
Lehrer debates:

http://groups.google.com/group/sci.med.diseases.lyme/browse_frm/thread/e4359868117b8d81/e066f6566802741e?q=lehrer+bush+gore+bombs+bursting+in+air&rnum=1#e066f6566802741e

BUSH:
"It's also important to keep strong ties in the Middle
East, credible ties, because of the energy crisis
we're now in. After all, a lot of the energy is
produced from the Middle East."

BUSH: Well, I think -- it's hard to tell. I think
that, you know, ***I would hope to be able to convince
people I could handle the Iraqi situation better. I
mean, we don't...

LEHRER: Saddam Hussein, you mean? ***

BUSH: Yes.

LEHRER: You could get him out of there?

BUSH: I'd like to, of course. And I presume this
administration would as well. But we don't know.
There's no inspectors now in Iraq. The coalition that
was in place isn't as strong as it used to be.

He is a danger.

BUSH: We don't want him fishing in troubled waters in
the Middle East. And it's going to be hard to -- it's
going to be important to rebuild that coalition to
keep the pressure on him.

LEHRER: Do you feel that is a failure of the Clinton
administration?

BUSH: I do.

GORE: Well, when I got to be a part of the current
administration, it was right after I was one of the
few
members of my political party to support former
President Bush in the Persian Gulf War resolution.

And at the end of that war, for whatever reasons, it
was not finished in a way that removed Saddam Hussein
from power. ***I know there are all kinds of
circumstances and explanations.***

But the fact is that that's the situation that was
left when I got there. And we have maintained the
sanctions."

---

Comment: George the Father left Saddam in
power, but it's Clinton's fault for not
resolving the issue. And George Junior
believes we need Arab Oil, so he'll "handle"
things.

Yeah, right. I can see it now...

"And so proudly we hail, the bombs bursting in air"
or however it goes.

It shall be glorious, I'm sure.

[Oct 2000, before he wasn't elected the *first* time.]
=====================================
The circumstances were the bioweapons we found out
that we sold Iraq earlier to use against Iran
resulting in Gulf War Illness, Chronic Fatigue
Syndrome, etc or whatever is today's flavor of Simon
Wessley.

Dealin wid duh UK gubment hos:
http://cvi.asm.org/cgi/content/full/6/1/6/T2 The
scientific validation of Simon Wessely's astuteness on
hypochondria as the cause of Gulf War Illness,
Medically Unexplained Persisting Symptoms (MUPS), and
Chronic Fatigue Syndrome.

(fairy alert)

Thanks Wessely. And NASA is holding on line 2 for
you.

http://bmj.bmjjournals.com/cgi/content/full/313/7072/1594
Psychological Medicine: That's where you tell sick
people they're liars and then pat yourself on the
back. It's actually where the bioweaponeers (stealth
fungal infections) pat themselves on the back for the
usefulness of such egotistical fools as Simon Wessely.
I can't wait to hear about the penis matters involved
in all those Amazonian frogs dying off from new fungi
!!
http://www.google.com/search?hl=en&q=frogs+amazon+fungi&btnG=Google+Search

Munchausens or Catastrophizing in frogs?
http://actionlyme.org/HOWE_BBC_SCIENCE_EVIDENCE.htm

Or is someone putting ticks on frogs like McSweegan
suggests?
http://actionlyme.org/MCSWEEGAN_AND_MUNCHAUSENS.htm

And these raving lunatics want to run the planet?

Kathleen M. Dickson
23 Garden Street
Pawcatuck, CT 06379
http://actionlyme.org

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